Search results for "Bioquímica clínica"

showing 5 items of 5 documents

Curcumin as a therapeutic option in retinal diseases

2020

Este artículo se encuentra disponible en la siguiente URL: https://www.mdpi.com/2076-3921/9/1/48 Este artículo pertenece al número especial "Natural products in health promotion and disease prevention". En este artículo también participa: Vincent M. Villar. The retina is subjected to oxidative stress due to its high vascularization, long time light exposition and a high density of mitochondria. Oxidative stress can lead to pathological processes, like cell apoptosis, angiogenesis and inflammation ending in retinal pathologies. Curcumin, a major bioactive component obtained from the spice turmeric (Curcuma longa) rhizome has been used for centuries in Asian countries for cooking and for curi…

0301 basic medicineBioquímicaretinaAntioxidantPhysiologyBioquímica clínicamedicine.medical_treatmentClinical BiochemistryCurcumina - Uso terapéutico.InflammationReviewPharmacologyMitochondrionmedicine.disease_causeBiochemistryRetina03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineoxidative stresscurcuminCurcumaMolecular BiologyVistachemistry.chemical_classificationEstrés oxidativo.Reactive oxygen speciesBiología molecularbiologybusiness.industryRetina - Diseases - Treatment.lcsh:RM1-950RetinalCell Biologybiology.organism_classificationCurcumin - Therapeutic use.Oxidative stress.030104 developmental biologylcsh:Therapeutics. Pharmacologyretinal diseaseschemistry030220 oncology & carcinogenesisCurcuminmedicine.symptombusinessCúrcumaOxidative stressRetina - Enfermedades - Tratamiento.
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Role of Ca2+-activated K+ channels and Na+,K+-ATPase in prostaglandin E1- and E2-induced inhibition of the adrenergic response in human vas deferens

2011

We studied the role of K(+) channels and Na(+),K(+)-ATPase in the presynaptic inhibitory effects of prostaglandin E(1) (PGE(1)) and PGE(2) on the adrenergic responses of human vas deferens. Furthermore, we determined the effects of increasing extracellular K(+) concentrations ([K(+)](o)) and inhibition of Na(+),K(+)-ATPase on neurogenic and norepinephrine-induced contractile responses. Ring segments of the epididymal part of the vas deferens were taken from 45 elective vasectomies and mounted in organ baths for isometric recording of tension. The neuromodulatory effects of PGEs were tested in the presence of K(+) channel blockers. PGE(1) and PGE(2) (10(-8) to 10(-6)M) induced inhibition of …

AdultMalemedicine.medical_specialtyBioquímica clínicaAdrenergicApaminSynaptic TransmissionBiochemistryDinoprostoneOuabainPotassium Channels Calcium-Activatedchemistry.chemical_compoundOrgan Culture TechniquesVas DeferensInternal medicinePotassium Channel BlockersmedicineHumansAlprostadilNa+/K+-ATPasePharmacologyDose-Response Relationship DrugChemistryVas deferensPotassium channel blockerIberiotoxinElectric StimulationPotassium channelReceptors AdrenergicEndocrinologymedicine.anatomical_structurelipids (amino acids peptides and proteins)Sodium-Potassium-Exchanging ATPaseMuscle Contractionmedicine.drugBiochemical Pharmacology
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Metabolomics-based strategy to assess drug hepatotoxicity and uncover the mechanisms of hepatotoxicity involved

2023

AbstractToxicity studies, among them hepatotoxicity, are key throughout preclinical stages of drug development to minimise undesired toxic effects that might eventually appear in the course of the clinical use of the new drug. Understanding the mechanism of injury of hepatotoxins is essential to efficiently anticipate their potential risk of toxicity in humans. The use of in vitro models and particularly cultured hepatocytes represents an easy and robust alternative to animal drug hepatotoxicity testing for predicting human risk. Here, we envisage an innovative strategy to identify potential hepatotoxic drugs, quantify the magnitude of the alterations caused, and uncover the mechanisms of t…

Bioquímica clínicaHealth Toxicology and MutagenesisGeneral MedicineToxicologyArchives of Toxicology
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Applying extracellular vesicles based therapeutics in clinical trials - an ISEV position paper.

2015

Extracellular vesicles (EVs), such as exosomes and microvesicles, are released by different cell types and participate in physiological and pathophysiological processes. EVs mediate intercellular communication as cell-derived extracellular signalling organelles that transmit specific information from their cell of origin to their target cells. As a result of these properties, EVs of defined cell types may serve as novel tools for various therapeutic approaches, including (a) anti-tumour therapy, (b) pathogen vaccination, (c) immune-modulatory and regenerative therapies and (d) drug delivery. The translation of EVs into clinical therapies requires the categorization of EV-based therapeutics …

Bioquímica clínicaMedizinISCHEMIA-REPERFUSION INJURYBioinformaticsimmunology; neurobiology; haematology; stem cells; tissue regeneration; tumour vaccination; regulationimmunology0302 clinical medicineClinical trialsClinical investigationVERSUS-HOST-DISEASEMedicine and Health SciencesFIELD-FLOW FRACTIONATIONMedicineImmunologiahaematology; immunology; neurobiology; regulation; stem cells; tissue regeneration; tumour vaccinationmedia_common0303 health scienceslcsh:CytologyOUTER-MEMBRANE VESICLESneurobiologyregulationHematologyBiologia experimental3. Good healthTUMOR-DERIVED EXOSOMES030220 oncology & carcinogenesistumour vaccinationDrug deliveryhaematologyPosition PaperCèl·lules mareNeurobiologiaHistologyMedicina InvestigacióCèl·lulesNANOPARTICLE TRACKING ANALYSIStissue regenerationExtracellular vesiclesMESENCHYMAL STEM-CELLS03 medical and health sciencesstem cellsJournal Articlemedia_common.cataloged_instanceREGULATORY T-CELLSEuropean unionlcsh:QH573-671ENDOTHELIAL PROGENITOR CELLSHematologia030304 developmental biologybusiness.industryCell BiologyMicrovesiclesClinical trialPosition paperPharmaceutical manufacturingUMBILICAL-CORD BLOODbusinessNeuroscienceAssaigs clínics
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Methionine Cycle Rewiring by Targeting miR-873-5p Modulates Ammonia Metabolism to Protect the Liver from Acetaminophen

2022

Drug-induced liver injury (DILI) development is commonly associated with acetaminophen (APAP) overdose, where glutathione scavenging leads to mitochondrial dysfunction and hepatocyte death. DILI is a severe disorder without effective late-stage treatment, since N-acetyl cysteine must be administered 8 h after overdose to be efficient. Ammonia homeostasis is altered during liver diseases and, during DILI, it is accompanied by decreased glycine N-methyltransferase (GNMT) expression and S-adenosylmethionine (AdoMet) levels that suggest a reduced methionine cycle. Anti-miR-873-5p treatment prevents cell death in primary hepatocytes and the appearance of necrotic areas in liver from APAP-adminis…

drug-induced liver injury (DILI); acetaminophen (APAP); ammonia; methionine cycle; miR-873-5p; therapy; polyamines; mitochondriatherapyacetaminophen (APAP)Bioquímica clínicaPhysiologypolyaminesClinical Biochemistrydrug-induced liver injury (DILI)Cell BiologyammoniamiR-873-5pBiochemistrymitochondriaParacetamolmethionine cycleMolecular BiologyAntioxidants
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